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{{html}}<hr style="border: 3px solid red;">{{/html}} |
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+💥 If this works, we can move on to the next study! 🚀 Let me know how it looks! |
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+I'll process the next study and populate the template accordingly. Let me extract the key details from the uploaded document now. |
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+Here's the structured summary for the next study: |
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## **📄 Download Full Study** |
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-[[Download Full Study>>attach:10.1007_s12144-023-04275-z.pdf]] |
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+{{velocity}} |
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+#set($doi = "10.1007_s12144-023-04275-z") |
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+#set($filename = "${doi}.pdf") |
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+#if($xwiki.exists("attach:$filename")) |
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+[[Download>>attach:$filename]] |
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+#else |
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+{{html}}<span style="color: red; font-weight: bold;">🚨 PDF Not Available 🚨</span>{{/html}} |
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+#end |
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+{{/velocity}} |
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{{html}}<hr style="border: 3px solid red;">{{/html}} |
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-{{expand title="Study: Associations Between Cannabis Use and Mental Health Symptoms in Young Adults" expanded="false"}} Source: Addictive Behaviors |
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-Date of Publication: 2016 |
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-Author(s): Andrea Hussong, Christy Capron, Gregory T. Smith, Jennifer L. Maggs |
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-Title: "Associations Between Cannabis Use and Mental Health Symptoms in Young Adults" |
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-DOI: 10.1016/j.addbeh.2016.02.030 |
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-Subject Matter: Substance Use, Mental Health, Adolescent Development |
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-Key Statistics |
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-General Observations: |
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-Study examined cannabis use trends in young adults over time. |
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-Found significant correlations between cannabis use and increased depressive symptoms. |
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-Subgroup Analysis: |
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- |
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-Males exhibited higher rates of cannabis use, but females reported stronger mental health impacts. |
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-Individuals with pre-existing anxiety disorders were more likely to report problematic cannabis use. |
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-Other Significant Data Points: |
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-Frequent cannabis users showed a 23% higher likelihood of developing anxiety symptoms. |
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-Co-occurring substance use (e.g., alcohol) exacerbated negative psychological effects. |
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-Findings |
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-Primary Observations: |
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-Cannabis use was linked to higher depressive and anxiety symptoms, particularly in frequent users. |
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-Self-medication patterns emerged among those with pre-existing mental health conditions. |
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-Subgroup Trends: |
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- |
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-Early cannabis initiation (before age 16) was associated with greater mental health risks. |
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-College-aged users reported more impairments in daily functioning due to cannabis use. |
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-Specific Case Analysis: |
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- |
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-Participants with a history of childhood trauma were twice as likely to develop problematic cannabis use. |
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-Co-use of cannabis and alcohol significantly increased impulsivity scores in the study sample. |
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-Critique and Observations |
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-Strengths of the Study: |
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- |
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-Large, longitudinal dataset with a diverse sample of young adults. |
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-Controlled for confounding variables like socioeconomic status and prior substance use. |
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-Limitations of the Study: |
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- |
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-Self-reported cannabis use may introduce bias in reported frequency and effects. |
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-Did not assess specific THC potency levels, which could influence mental health outcomes. |
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-Suggestions for Improvement: |
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- |
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-Future research should investigate dose-dependent effects of cannabis on mental health. |
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-Assess long-term psychological outcomes of early cannabis exposure. |
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-Relevance to Subproject |
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-Supports mental health risk assessment models related to substance use. |
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-Highlights gender differences in substance-related psychological impacts. |
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-Provides insight into self-medication behaviors among young adults. |
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-Suggestions for Further Exploration |
360 |
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-Investigate the long-term impact of cannabis use on neurodevelopment. |
361 |
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-Examine the role of genetic predisposition in cannabis-related mental health risks. |
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-Assess regional differences in cannabis use trends post-legalization. |
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-Summary of Research Study |
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-This study examines the relationship between cannabis use and mental health symptoms in young adults, focusing on depressive and anxiety-related outcomes. Using a longitudinal dataset, the researchers found higher risks of anxiety and depression in frequent cannabis users, particularly among those with pre-existing mental health conditions or early cannabis initiation. |
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-This summary provides an accessible, at-a-glance overview of the study’s contributions. Please refer to the full paper for in-depth analysis. |
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- |
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-📄 Download Full Study |
369 |
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-[[Download Full Study>>attach:10.1016_j.addbeh.2016.02.030.pdf]] |
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-{{/expand}} |
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-{{html}}<hr style="border: 3px solid red;">{{/html}} |
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-{{expand title="Study: Is there a Dysgenic Secular Trend Towards Slowing Simple Reaction Time?" expanded="false"}} |
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-**Source:** *Intelligence (Elsevier)* |
377 |
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-**Date of Publication:** *2014* |
378 |
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-**Author(s):** *Michael A. Woodley, Jan te Nijenhuis, Raegan Murphy* |
379 |
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-**Title:** *"Is there a Dysgenic Secular Trend Towards Slowing Simple Reaction Time?"* |
380 |
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-**DOI:** [10.1016/j.intell.2014.05.012](https://doi.org/10.1016/j.intell.2014.05.012) |
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-**Subject Matter:** *Cognitive Decline, Intelligence, Dysgenics* |
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- |
383 |
|
---- |
384 |
|
- |
385 |
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-## **Key Statistics** |
386 |
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-1. **General Observations:** |
387 |
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- - The study examines reaction time data from **13 age-matched studies** spanning **1884–2004**. |
388 |
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- - Results suggest an estimated **decline of 13.35 IQ points** over this period. |
389 |
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- |
390 |
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-2. **Subgroup Analysis:** |
391 |
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- - The study found **slower reaction times in modern populations** compared to Victorian-era individuals. |
392 |
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- - Data from **Western countries (US, UK, Canada, Australia, Finland)** were analyzed. |
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- |
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-3. **Other Significant Data Points:** |
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- - The estimated **dysgenic rate is 1.21 IQ points lost per decade**. |
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- - Meta-regression analysis confirmed a **steady secular trend in slowing reaction time**. |
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- |
398 |
|
---- |
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- |
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-## **Findings** |
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-1. **Primary Observations:** |
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- - Supports the hypothesis of **intelligence decline due to genetic and environmental factors**. |
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- - Reaction time, a **biomarker for cognitive ability**, has slowed significantly over time. |
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- |
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-2. **Subgroup Trends:** |
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- - A stronger **correlation between slower reaction time and lower general intelligence (g)**. |
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- - Flynn effect (IQ gains) does not contradict this finding, as reaction time is a **biological, not environmental, measure**. |
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- |
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-3. **Specific Case Analysis:** |
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- - Cross-national comparisons indicate a **global trend in slower reaction times**. |
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- - Factors like **modern neurotoxin exposure** and **reduced selective pressure for intelligence** may contribute. |
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- |
413 |
|
---- |
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- |
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-## **Critique and Observations** |
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-1. **Strengths of the Study:** |
417 |
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- - **Comprehensive meta-analysis** covering over a century of reaction time data. |
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- - **Robust statistical corrections** for measurement variance between historical and modern studies. |
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- |
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-2. **Limitations of the Study:** |
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- - Some historical data sources **lack methodological consistency**. |
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- - **Reaction time measurements vary by study**, requiring adjustments for equipment differences. |
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- |
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-3. **Suggestions for Improvement:** |
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- - Future studies should **replicate results with more modern datasets**. |
426 |
|
- - Investigate **alternative cognitive biomarkers** for intelligence over time. |
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- |
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|
---- |
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- |
430 |
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-## **Relevance to Subproject** |
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-- Provides evidence for **long-term intelligence trends**, contributing to research on **cognitive evolution**. |
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-- Aligns with broader discussions on **dysgenics, neurophysiology, and cognitive load**. |
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-- Supports the argument that **modern societies may be experiencing intelligence decline**. |
434 |
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- |
435 |
|
---- |
436 |
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- |
437 |
|
-## **Suggestions for Further Exploration** |
438 |
|
-1. Investigate **genetic markers associated with reaction time** and intelligence decline. |
439 |
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-2. Examine **regional variations in reaction time trends**. |
440 |
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-3. Explore **cognitive resilience factors that counteract the decline**. |
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|
- |
442 |
|
---- |
443 |
|
- |
444 |
|
-## **Summary of Research Study** |
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|
-This study examines **historical reaction time data** as a measure of **cognitive ability and intelligence decline**, analyzing data from **Western populations between 1884 and 2004**. The results suggest a **measurable decline in intelligence, estimated at 13.35 IQ points**, likely due to **dysgenic fertility, neurophysiological factors, and reduced selection pressures**. |
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- |
447 |
|
-This summary provides an accessible, at-a-glance overview of the study’s contributions. Please refer to the full paper for in-depth analysis. |
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- |
449 |
|
---- |
450 |
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- |
451 |
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-## **📄 Download Full Study** |
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-[[Download Full Study>>attach:10.1016_j.intell.2014.05.012.pdf]] |
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-{{/expand}} |
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-{{html}}<hr style="border: 3px solid red;">{{/html}} |
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-{{expand title="Study: A Review of Intelligence GWAS Hits: Their Relationship to Country IQ and the Issue of Spatial Autocorrelation" expanded="false"}} |
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-**Source:** *Intelligence (Elsevier)* |
460 |
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-**Date of Publication:** *2015* |
461 |
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-**Author(s):** *Davide Piffer* |
462 |
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-**Title:** *"A Review of Intelligence GWAS Hits: Their Relationship to Country IQ and the Issue of Spatial Autocorrelation"* |
463 |
|
-**DOI:** [10.1016/j.intell.2015.08.008](https://doi.org/10.1016/j.intell.2015.08.008) |
464 |
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-**Subject Matter:** *Genetics, Intelligence, GWAS, Population Differences* |
465 |
|
- |
466 |
|
---- |
467 |
|
- |
468 |
|
-## **Key Statistics** |
469 |
|
-1. **General Observations:** |
470 |
|
- - Study analyzed **genome-wide association studies (GWAS) hits** linked to intelligence. |
471 |
|
- - Found a **strong correlation (r = .91) between polygenic intelligence scores and national IQ levels**. |
472 |
|
- |
473 |
|
-2. **Subgroup Analysis:** |
474 |
|
- - Factor analysis of **9 intelligence-associated alleles** revealed a metagene correlated with **country IQ (r = .86)**. |
475 |
|
- - **Allele frequencies varied significantly by continent**, aligning with observed population differences in cognitive ability. |
476 |
|
- |
477 |
|
-3. **Other Significant Data Points:** |
478 |
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- - GWAS intelligence SNPs predicted **IQ levels more strongly than random genetic markers**. |
479 |
|
- - Genetic differentiation (Fst values) showed that **selection pressure, rather than drift, influenced intelligence-related allele distributions**. |
480 |
|
- |
481 |
|
---- |
482 |
|
- |
483 |
|
-## **Findings** |
484 |
|
-1. **Primary Observations:** |
485 |
|
- - Intelligence-associated SNP frequencies correlate **highly with national IQ levels**. |
486 |
|
- - Genetic selection for intelligence appears **stronger than selection for height-related genes**. |
487 |
|
- |
488 |
|
-2. **Subgroup Trends:** |
489 |
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- - **East Asian populations** exhibited the **highest frequencies of intelligence-associated alleles**. |
490 |
|
- - **African populations** showed lower frequencies compared to European and East Asian populations. |
491 |
|
- |
492 |
|
-3. **Specific Case Analysis:** |
493 |
|
- - Polygenic scores using **intelligence-related alleles significantly outperformed random SNPs** in predicting IQ. |
494 |
|
- - Selection pressures **may explain differences in global intelligence distribution** beyond genetic drift effects. |
495 |
|
- |
496 |
|
---- |
497 |
|
- |
498 |
|
-## **Critique and Observations** |
499 |
|
-1. **Strengths of the Study:** |
500 |
|
- - **Comprehensive genetic analysis** of intelligence-linked SNPs. |
501 |
|
- - Uses **multiple statistical methods (factor analysis, Fst analysis) to confirm results**. |
502 |
|
- |
503 |
|
-2. **Limitations of the Study:** |
504 |
|
- - **Correlation does not imply causation**; factors beyond genetics influence intelligence. |
505 |
|
- - **Limited number of GWAS-identified intelligence alleles**—future studies may identify more. |
506 |
|
- |
507 |
|
-3. **Suggestions for Improvement:** |
508 |
|
- - Larger **cross-population GWAS studies** needed to validate findings. |
509 |
|
- - Investigate **non-genetic contributors to IQ variance** in addition to genetic factors. |
510 |
|
- |
511 |
|
---- |
512 |
|
- |
513 |
|
-## **Relevance to Subproject** |
514 |
|
-- Supports research on **genetic influences on intelligence at a population level**. |
515 |
|
-- Aligns with broader discussions on **cognitive genetics and natural selection effects**. |
516 |
|
-- Provides a **quantitative framework for analyzing polygenic selection in intelligence studies**. |
517 |
|
- |
518 |
|
---- |
519 |
|
- |
520 |
|
-## **Suggestions for Further Exploration** |
521 |
|
-1. Conduct **expanded GWAS studies** including diverse populations. |
522 |
|
-2. Investigate **gene-environment interactions influencing intelligence**. |
523 |
|
-3. Explore **historical selection pressures shaping intelligence-related alleles**. |
524 |
|
- |
525 |
|
---- |
526 |
|
- |
527 |
|
-## **Summary of Research Study** |
528 |
|
-This study reviews **genome-wide association study (GWAS) findings on intelligence**, demonstrating a **strong correlation between polygenic intelligence scores and national IQ levels**. The research highlights how **genetic selection may explain population-level cognitive differences beyond genetic drift effects**. Intelligence-linked alleles showed **higher variability across populations than height-related alleles**, suggesting stronger selection pressures. |
529 |
|
- |
530 |
|
-This summary provides an accessible, at-a-glance overview of the study’s contributions. Please refer to the full paper for in-depth analysis. |
531 |
|
- |
532 |
|
---- |
533 |
|
- |
534 |
|
-## **📄 Download Full Study** |
535 |
|
-[[Download Full Study>>attach:10.1016_j.intell.2015.08.008.pdf]] |
536 |
|
- |
537 |
|
-{{/expand}} |
538 |
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- |
539 |
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-{{html}}<hr style="border: 3px solid red;">{{/html}} |
540 |
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- |
541 |
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-{{expand title="Study: Survey of Expert Opinion on Intelligence: Intelligence Research, Experts' Background, Controversial Issues, and the Media" expanded="false"}} |
542 |
|
-**Source:** *Intelligence (Elsevier)* |
543 |
|
-**Date of Publication:** *2019* |
544 |
|
-**Author(s):** *Heiner Rindermann, David Becker, Thomas R. Coyle* |
545 |
|
-**Title:** *"Survey of Expert Opinion on Intelligence: Intelligence Research, Experts' Background, Controversial Issues, and the Media"* |
546 |
|
-**DOI:** [10.1016/j.intell.2019.101406](https://doi.org/10.1016/j.intell.2019.101406) |
547 |
|
-**Subject Matter:** *Psychology, Intelligence Research, Expert Analysis* |
548 |
|
- |
549 |
|
---- |
550 |
|
- |
551 |
|
-## **Key Statistics** |
552 |
|
-1. **General Observations:** |
553 |
|
- - Survey of **102 experts** on intelligence research and public discourse. |
554 |
|
- - Evaluated experts' backgrounds, political affiliations, and views on controversial topics in intelligence research. |
555 |
|
- |
556 |
|
-2. **Subgroup Analysis:** |
557 |
|
- - **90% of experts were from Western countries**, and **83% were male**. |
558 |
|
- - Political spectrum ranged from **54% left-liberal, 24% conservative**, with significant ideological influences on views. |
559 |
|
- |
560 |
|
-3. **Other Significant Data Points:** |
561 |
|
- - Experts rated media coverage of intelligence research as **poor (avg. 3.1 on a 9-point scale)**. |
562 |
|
- - **50% of experts attributed US Black-White IQ differences to genetic factors, 50% to environmental factors**. |
563 |
|
- |
564 |
|
---- |
565 |
|
- |
566 |
|
-## **Findings** |
567 |
|
-1. **Primary Observations:** |
568 |
|
- - Experts overwhelmingly support **the g-factor theory of intelligence**. |
569 |
|
- - **Heritability of intelligence** was widely accepted, though views differed on race and group differences. |
570 |
|
- |
571 |
|
-2. **Subgroup Trends:** |
572 |
|
- - **Left-leaning experts were more likely to reject genetic explanations for group IQ differences**. |
573 |
|
- - **Right-leaning experts tended to favor a stronger role for genetic factors** in intelligence disparities. |
574 |
|
- |
575 |
|
-3. **Specific Case Analysis:** |
576 |
|
- - The study compared **media coverage of intelligence research** with expert opinions. |
577 |
|
- - Found a **disconnect between journalists and intelligence researchers**, especially regarding politically sensitive issues. |
578 |
|
- |
579 |
|
---- |
580 |
|
- |
581 |
|
-## **Critique and Observations** |
582 |
|
-1. **Strengths of the Study:** |
583 |
|
- - **Largest expert survey on intelligence research** to date. |
584 |
|
- - Provides insight into **how political orientation influences scientific perspectives**. |
585 |
|
- |
586 |
|
-2. **Limitations of the Study:** |
587 |
|
- - **Sample primarily from Western countries**, limiting global perspectives. |
588 |
|
- - Self-selection bias may skew responses toward **those more willing to engage with controversial topics**. |
589 |
|
- |
590 |
|
-3. **Suggestions for Improvement:** |
591 |
|
- - Future studies should include **a broader range of global experts**. |
592 |
|
- - Additional research needed on **media biases and misrepresentation of intelligence research**. |
593 |
|
- |
594 |
|
---- |
595 |
|
- |
596 |
|
-## **Relevance to Subproject** |
597 |
|
-- Provides insight into **expert consensus and division on intelligence research**. |
598 |
|
-- Highlights the **role of media bias** in shaping public perception of intelligence science. |
599 |
|
-- Useful for understanding **the intersection of science, politics, and public discourse** on intelligence research. |
600 |
|
- |
601 |
|
---- |
602 |
|
- |
603 |
|
-## **Suggestions for Further Exploration** |
604 |
|
-1. Examine **cross-national differences** in expert opinions on intelligence. |
605 |
|
-2. Investigate how **media bias impacts public understanding of intelligence research**. |
606 |
|
-3. Conduct follow-up studies with **a more diverse expert pool** to test findings. |
607 |
|
- |
608 |
|
---- |
609 |
|
- |
610 |
|
-## **Summary of Research Study** |
611 |
|
-This study surveys **expert opinions on intelligence research**, analyzing **how backgrounds, political ideologies, and media representation influence perspectives on intelligence**. The findings highlight **divisions in scientific consensus**, particularly on **genetic vs. environmental causes of IQ disparities**. Additionally, the research uncovers **widespread dissatisfaction with media portrayals of intelligence research**, pointing to **the impact of ideological biases on public discourse**. |
612 |
|
- |
613 |
|
-This summary provides an accessible, at-a-glance overview of the study’s contributions. Please refer to the full paper for in-depth analysis. |
614 |
|
- |
615 |
|
---- |
616 |
|
- |
617 |
|
-## **📄 Download Full Study** |
618 |
|
-[[Download Full Study>>attach:10.1016_j.intell.2019.101406.pdf]] |
619 |
|
- |
620 |
|
-{{/expand}} |
621 |
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- |
622 |
|
-{{html}}<hr style="border: 3px solid red;">{{/html}} |
623 |
|
- |
624 |
|
-{{expand title="Study: Is Homo sapiens polytypic? Human taxonomic diversity and its implications" expanded="false"}} |
625 |
|
-**Source:** *Medical Hypotheses (Elsevier)* |
626 |
|
-**Date of Publication:** *2010* |
627 |
|
-**Author(s):** *Michael A. Woodley* |
628 |
|
-**Title:** *"Is Homo sapiens polytypic? Human taxonomic diversity and its implications"* |
629 |
|
-**DOI:** [10.1016/j.mehy.2009.07.046](https://doi.org/10.1016/j.mehy.2009.07.046) |
630 |
|
-**Subject Matter:** *Human Taxonomy, Evolutionary Biology, Anthropology* |
631 |
|
- |
632 |
|
---- |
633 |
|
- |
634 |
|
-## **Key Statistics** |
635 |
|
-1. **General Observations:** |
636 |
|
- - The study argues that **Homo sapiens is polytypic**, meaning it consists of multiple subspecies rather than a single monotypic species. |
637 |
|
- - Examines **genetic diversity, morphological variation, and evolutionary lineage** in humans. |
638 |
|
- |
639 |
|
-2. **Subgroup Analysis:** |
640 |
|
- - Discusses **four primary definitions of race/subspecies**: Essentialist, Taxonomic, Population-based, and Lineage-based. |
641 |
|
- - Suggests that **human heterozygosity levels are comparable to species that are classified as polytypic**. |
642 |
|
- |
643 |
|
-3. **Other Significant Data Points:** |
644 |
|
- - The study evaluates **FST values (genetic differentiation measure)** and argues that human genetic differentiation is comparable to that of recognized subspecies in other species. |
645 |
|
- - Considers **phylogenetic species concepts** in defining human variation. |
646 |
|
- |
647 |
|
---- |
648 |
|
- |
649 |
|
-## **Findings** |
650 |
|
-1. **Primary Observations:** |
651 |
|
- - Proposes that **modern human populations meet biological criteria for subspecies classification**. |
652 |
|
- - Highlights **medical and evolutionary implications** of human taxonomic diversity. |
653 |
|
- |
654 |
|
-2. **Subgroup Trends:** |
655 |
|
- - Discusses **how race concepts evolved over time** in biological sciences. |
656 |
|
- - Compares **human diversity with that of other primates** such as chimpanzees and gorillas. |
657 |
|
- |
658 |
|
-3. **Specific Case Analysis:** |
659 |
|
- - Evaluates how **genetic markers correlate with population structure**. |
660 |
|
- - Addresses the **controversy over race classification in modern anthropology**. |
661 |
|
- |
662 |
|
---- |
663 |
|
- |
664 |
|
-## **Critique and Observations** |
665 |
|
-1. **Strengths of the Study:** |
666 |
|
- - Uses **comparative species analysis** to assess human classification. |
667 |
|
- - Provides a **biological perspective** on the race concept, moving beyond social constructivism arguments. |
668 |
|
- |
669 |
|
-2. **Limitations of the Study:** |
670 |
|
- - Controversial topic with **strong opposing views in anthropology and genetics**. |
671 |
|
- - **Relies on broad genetic trends**, but does not analyze individual-level genetic variation in depth. |
672 |
|
- |
673 |
|
-3. **Suggestions for Improvement:** |
674 |
|
- - Further research should **incorporate whole-genome studies** to refine subspecies classifications. |
675 |
|
- - Investigate **how admixture affects taxonomic classification over time**. |
676 |
|
- |
677 |
|
---- |
678 |
|
- |
679 |
|
-## **Relevance to Subproject** |
680 |
|
-- Contributes to discussions on **evolutionary taxonomy and species classification**. |
681 |
|
-- Provides evidence on **genetic differentiation among human populations**. |
682 |
|
-- Highlights **historical and contemporary scientific debates on race and human variation**. |
683 |
|
- |
684 |
|
---- |
685 |
|
- |
686 |
|
-## **Suggestions for Further Exploration** |
687 |
|
-1. Examine **FST values in modern and ancient human populations**. |
688 |
|
-2. Investigate how **adaptive evolution influences population differentiation**. |
689 |
|
-3. Explore **the impact of genetic diversity on medical treatments and disease susceptibility**. |
690 |
|
- |
691 |
|
---- |
692 |
|
- |
693 |
|
-## **Summary of Research Study** |
694 |
|
-This study evaluates **whether Homo sapiens should be classified as a polytypic species**, analyzing **genetic diversity, evolutionary lineage, and morphological variation**. Using comparative analysis with other primates and mammals, the research suggests that **human populations meet biological criteria for subspecies classification**, with implications for **evolutionary biology, anthropology, and medicine**. |
695 |
|
- |
696 |
|
-This summary provides an accessible, at-a-glance overview of the study’s contributions. Please refer to the full paper for in-depth analysis. |
697 |
|
- |
698 |
|
---- |
699 |
|
- |
700 |
|
-## **📄 Download Full Study** |
701 |
|
-[[Download Full Study>>attach:10.1016_j.mehy.2009.07.046.pdf]] |
702 |
|
- |
703 |
|
-{{/expand}} |
704 |
|
- |
705 |
|
-{{html}}<hr style="border: 3px solid red;">{{/html}} |
706 |
|
- |
707 |
|
-{{expand title="Study: The Wilson Effect: The Increase in Heritability of IQ With Age" expanded="false"}} |
708 |
|
-**Source:** *Twin Research and Human Genetics (Cambridge University Press)* |
709 |
|
-**Date of Publication:** *2013* |
710 |
|
-**Author(s):** *Thomas J. Bouchard Jr.* |
711 |
|
-**Title:** *"The Wilson Effect: The Increase in Heritability of IQ With Age"* |
712 |
|
-**DOI:** [10.1017/thg.2013.54](https://doi.org/10.1017/thg.2013.54) |
713 |
|
-**Subject Matter:** *Intelligence, Heritability, Developmental Psychology* |
714 |
|
- |
715 |
|
---- |
716 |
|
- |
717 |
|
-## **Key Statistics** |
718 |
|
-1. **General Observations:** |
719 |
|
- - The study documents how the **heritability of IQ increases with age**, reaching an asymptote at **0.80 by adulthood**. |
720 |
|
- - Analysis is based on **longitudinal twin and adoption studies**. |
721 |
|
- |
722 |
|
-2. **Subgroup Analysis:** |
723 |
|
- - Shared environmental influence on IQ **declines with age**, reaching **0.10 in adulthood**. |
724 |
|
- - Monozygotic twins show **increasing genetic similarity in IQ over time**, while dizygotic twins become **less concordant**. |
725 |
|
- |
726 |
|
-3. **Other Significant Data Points:** |
727 |
|
- - Data from the **Louisville Longitudinal Twin Study and cross-national twin samples** support findings. |
728 |
|
- - IQ stability over time is **influenced more by genetics than by shared environmental factors**. |
729 |
|
- |
730 |
|
---- |
731 |
|
- |
732 |
|
-## **Findings** |
733 |
|
-1. **Primary Observations:** |
734 |
|
- - Intelligence heritability **strengthens throughout development**, contrary to early environmental models. |
735 |
|
- - Shared environmental effects **decrease by late adolescence**, emphasizing **genetic influence in adulthood**. |
736 |
|
- |
737 |
|
-2. **Subgroup Trends:** |
738 |
|
- - Studies from **Scotland, Netherlands, and the US** show **consistent patterns of increasing heritability with age**. |
739 |
|
- - Findings hold across **varied socio-economic and educational backgrounds**. |
740 |
|
- |
741 |
|
-3. **Specific Case Analysis:** |
742 |
|
- - Longitudinal adoption studies show **declining impact of adoptive parental influence on IQ** as children age. |
743 |
|
- - Cross-sectional twin data confirm **higher IQ correlations for monozygotic twins in adulthood**. |
744 |
|
- |
745 |
|
---- |
746 |
|
- |
747 |
|
-## **Critique and Observations** |
748 |
|
-1. **Strengths of the Study:** |
749 |
|
- - **Robust dataset covering multiple twin and adoption studies over decades**. |
750 |
|
- - **Clear, replicable trend** demonstrating the increasing role of genetics in intelligence. |
751 |
|
- |
752 |
|
-2. **Limitations of the Study:** |
753 |
|
- - Findings apply primarily to **Western industrialized nations**, limiting generalizability. |
754 |
|
- - **Lack of neurobiological mechanisms** explaining how genes express their influence over time. |
755 |
|
- |
756 |
|
-3. **Suggestions for Improvement:** |
757 |
|
- - Future research should investigate **gene-environment interactions in cognitive aging**. |
758 |
|
- - Examine **heritability trends in non-Western populations** to determine cross-cultural consistency. |
759 |
|
- |
760 |
|
---- |
761 |
|
- |
762 |
|
-## **Relevance to Subproject** |
763 |
|
-- Provides **strong evidence for the genetic basis of intelligence**. |
764 |
|
-- Highlights the **diminishing role of shared environment in cognitive development**. |
765 |
|
-- Supports research on **cognitive aging and heritability across the lifespan**. |
766 |
|
- |
767 |
|
---- |
768 |
|
- |
769 |
|
-## **Suggestions for Further Exploration** |
770 |
|
-1. Investigate **neurogenetic pathways underlying IQ development**. |
771 |
|
-2. Examine **how education and socioeconomic factors interact with genetic IQ influences**. |
772 |
|
-3. Study **heritability trends in aging populations and cognitive decline**. |
773 |
|
- |
774 |
|
---- |
775 |
|
- |
776 |
|
-## **Summary of Research Study** |
777 |
|
-This study documents **The Wilson Effect**, demonstrating how the **heritability of IQ increases throughout development**, reaching a plateau of **0.80 by adulthood**. The findings indicate that **shared environmental effects diminish with age**, while **genetic influences on intelligence strengthen**. Using **longitudinal twin and adoption data**, the research provides **strong empirical support for the increasing role of genetics in cognitive ability over time**. |
778 |
|
- |
779 |
|
-This summary provides an accessible, at-a-glance overview of the study’s contributions. Please refer to the full paper for in-depth analysis. |
780 |
|
- |
781 |
|
---- |
782 |
|
- |
783 |
|
-## **📄 Download Full Study** |
784 |
|
-[[Download Full Study>>attach:10.1017_thg.2013.54.pdf]] |
785 |
|
- |
786 |
|
-{{/expand}} |
787 |
|
- |
788 |
|
-{{html}}<hr style="border: 3px solid red;">{{/html}} |